İnci, SinanAksan, GökhanDoğan, Ali Kenan13.07.20192019-07-1613.07.20192019-07-1620151302-8723https://dx.doi.org/10.5152/AnatolJCardiol.2015.6641https://hdl.handle.net/20.500.12451/2449PubMed ID: 26574765To our knowledge, this patient is the first bonsai-induced Kounis syndrome case in literature. Kounis syndrome, in other words allergic MI, has two types depending on the pathophysiology, or the presence of coronary artery disease. In type I, patients exhibit coronary vasospasms induced by allergic mediators such as histamine, thromboxane, and leukotrienes without the presence of atherosclerosis risk factors or coronary artery disease. In type 2, ACS develops due to coronary vasospasms, plaque erosion, or plaque rupture induced by these mediators in patients with atherosclerotic coronary artery disease. Recently, the fact that there are eosinophil and mast cells in the thrombus material excised from some patients in whom stent thrombosis developed after stent implantation with drug release makes us consider hypersensitivity reactions in these patients. This situation is accepted as the type III variant of Kounis Syndrome (4). With these findings, our case is in accordance with the type I variant of Kounis syndromeeninfo:eu-repo/semantics/openAccessBonsai-induced kounis syndrome in a young male patientOther151195295410.5152/AnatolJCardiol.2015.6641N/A